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Is Pregabalin addicitive

Sep 16, 2026 · 5 sources used · OpenNeedle synthesis
Yes, pregabalin is addictive. The evidence shows it causes a diagnosable use disorder, is misused recreationally, and produces withdrawal symptoms.

The strongest evidence comes from case reports and a systematic review. A 2024 case report describes a patient who escalated to 3000 mg/day and met criteria for pregabalin use disorder [1]. A 2023 report of two migrants in a detention center diagnosed them with moderate pregabalin use disorder, noting tolerance, craving, and withdrawal [2]. That same paper cites a 2021 systematic review that found 19 cases of pregabalin abuse, with 16 showing tolerance and withdrawal [2].

A 2020 review in the British Journal of Pain confirms that pregabalin has more abuse potential than gabapentin, and both were reclassified as class C controlled substances in the UK in 2019 [3]. A 2020 Australian poison center study of 488 pregabalin overdose presentations found that 121 (25%) were recreational exposures, and 107 patients had a history of substance use disorder [4]. A 2025 Libyan cross-sectional study of 80 pregabalin users found that 57.5% reported non-medical use [5].

The mechanism is straightforward. Pregabalin binds to the alpha-2-delta subunit of voltage-gated calcium channels, reducing neurotransmitter release. This produces euphoria, sedation, and a calming effect that people with a history of substance abuse find rewarding. Withdrawal symptoms include anxiety, insomnia, agitation, and in severe cases, seizures [2][3].

The risk is not equal for everyone. People with a history of substance use disorder, especially opioid or benzodiazepine dependence, are at much higher risk of misusing pregabalin [2][3][4]. But the drug can produce dependence even in people who start it for a legitimate medical reason like nerve pain or fibromyalgia.

My call: pregabalin is addictive, with documented cases of use disorder, recreational misuse, and withdrawal. The risk is highest in people with a history of substance abuse, but anyone taking it long-term can develop physical dependence. Confidence: high.

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Sources used 5

  1. Silencing the phantom - pregabalin misuse in amputation patients: a case report Journal of Dependence (2024) Thin

    A 25-year-old right-arm amputee developed pregabalin use disorder with doses escalating to 3000 mg/day after self-medicating phantom limb sensations, and improved with duloxetine, lorazepam, mirtazapine, and weekly psychotherapy.

    DOI: 10.51982/bagimli.1458224
  2. “Just give me pregabalin, doctor”: pregabalin use disorder in two undocumented migrants from North Africa Discover Psychology (2023) primary study Strong

    Two undocumented North African migrants in a Portuguese detention center presented with moderate pregabalin use disorder, highlighting the drug's abuse potential in vulnerable migrant populations.

    DOI: 10.1007/s44202-023-00093-3
  3. Gabapentinoids: pharmacokinetics, pharmacodynamics and considerations for clinical practice British Journal of Pain (2020) Thin

    This article reviews the pharmacokinetics, pharmacodynamics, clinical use, safety, and regulatory considerations of gabapentin and pregabalin in neuropathic and non-neuropathic pain, highlighting their mechanisms, efficacy, risks, and perioperative use.

    DOI: 10.1177/2049463720912496
  4. Pregabalin poisoning and rising recreational use: a retrospective observational series British Journal of Clinical Pharmacology (2020) Thin

    A five-year retrospective observational study of pregabalin poisoning at two Australian toxicology units (2014–2019) describing patient demographics, dosing, co-ingestants, clinical effects (coma, seizures, hypotension), outcomes, and a rising prevalence of recreational use, con…

    DOI: 10.1111/bcp.14348
  5. Pregabalin Misuse and Associated Adverse Events: A Cross-Sectional Study in Libya Libyan Journal of Medical Research (2025) Mixed

    In a single-center sample of 80 adult pregabalin users, 57.5% reported non-medical use; non-medical use was associated with nausea, while sleep disturbance showed no significant association.

    DOI: 10.54361/ljmr.19.2.38

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